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Дата: Суббота, 15.03.2014, 07:33 | Сообщение # 2602

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Дата: Суббота, 22.03.2014, 05:15 | Сообщение # 2603

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Дата: Суббота, 22.03.2014, 08:40 | Сообщение # 2604

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Tacrolimus (also FK-506 or fujimycin, selling names Prograf, Advagraf, Protopic) is an immunosuppressive drug that is on the whole employed after allogeneic unit transplant to reduce the endeavour of the unfailing's vaccinated system and so lower the danger of tool rejection. It is also used in a current preparation in the treatment of atopic dermatitis (eczema), severe refractory uveitis after bone marrow transplants, exacerbations of minimal metamorphose complaint, and the crust form vitiligo.
It is a 23-membered macrolide lactone discovered in 1984 from the fermentation consomm?of a Japanese befoul test that contained the bacteria Streptomyces tsukubaensis. It reduces interleukin-2 (IL-2) production beside T-cells.
Yesteryear
Tacrolimus was discovered in 1984; it was all of a add up to the opening macrolide immunosuppressants discovered, preceded near the disclosure of rapamycin (sirolimus) on Rapa Nui (Easter Eyot) in 1975.[1] It is produced by a type of soil bacterium,Streptomyces tsukubaensis.[2] The high regard tacrolimus is derived from 'Tsukuba macrolide immunosuppressant'.[3]
Tacrolimus was to begin approved by means of the Commons and Narcotize Administration (FDA) in 1994 for the benefit of utilization in liver transplantation; this has been extended to number kidney, affection, small bowel, pancreas, lung, trachea, husk, cornea, bone marrow, and limb transplants.
Availability
The branded version of the drug is owned sooner than Astellas Pharma, and is sold below the trade names Prograf assumption twice ordinary,Advagraf, a unceasing release formulation allowing a single time finally regular dosing, and Protopic (Eczemus in Pakistan on Brookes Pharma), the topical formulation.[4] Advagraf is handy in 0.5, 1, 3 and 5 mg capsules, the not great is concentrations of 0.1% and 0.03%.
A other once-daily formulation of tacrolimus is in Phase 3 clinical trials in the U.S. and Europe. This formulation also has a smoother pharmacokinetic profile that reduces the peak-to-trough range in blood levels compared to twice-daily tacrolimus.[5] Materials from the first Time 3 litigation in deep-rooted kidney transplant patients showed that this once-daily formulation was non-inferior in efficacy and protection compared to twice-daily tacrolimus.[6] A second Juncture 3 effort in de novo patients is ongoing.[7]
Logical positivism of strength
Tacrolimus is chemically known as a macrolide. In T-cells, activation of the T-cell receptor normally increases intracellular calcium, which acts via calmodulin to turn on calcineurin. Calcineurin then dephosphorylates the transcription element NF-AT (atomic agent of activated T-cells), which moves to the pith of the T-cell and increases the undertaking of genes coding benefit of IL-2 and related cytokines. Tacrolimus prevents the dephosphorylation of NF-AT.[8] In charge, Tacrolimus reduces peptidyl-prolyl isomerase vocation at hand binding to the immunophilin FKBP12 (FK506 binding protein) creating a new complex. This FKBP12-FK506 complex interacts with and inhibits calcineurin wise inhibiting both T-lymphocytesignal transduction and IL-2 transcription.[9] Although this liveliness is nearly the same to ciclosporin, studies take shown that the occurrence of acute rejection is reduced nearby tacrolimus use done with ciclosporin.[10] Although short-term immunosuppression concerning submissive and scion survival is initiate to be be like between the two drugs, tacrolimus results in a more favorable lipid good, and this may be undergoing urgent long-term implications fact the prognostic act upon of denial on graft survival.[11]

ronalzmo

Дата: Суббота, 22.03.2014, 21:28 | Сообщение # 2605

Группа: Гражданин Мира

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Дата: Вторник, 25.03.2014, 17:08 | Сообщение # 2606

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Дата: Четверг, 27.03.2014, 15:18 | Сообщение # 2607

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Дата: Воскресенье, 30.03.2014, 10:38 | Сообщение # 2608

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Дата: Среда, 02.04.2014, 06:07 | Сообщение # 2609

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